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UCHL1-C90A activates TGFB. 1 / 1

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"Overexpression of UCH-L1, but not of UCH-L3 (the other human homolog of UBH1) or of the catalytic mutant UCH-L1 C90A, enhanced TGF-beta and SMAD-induced transcriptional activity, indicating that the deubiquitination activity of UCH-L1 is indispensable for enhancing TGF-beta and SMAD signaling."

33516505

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IndraLab

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UCHL1-C90A activates TGFB. 1 / 1

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