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This Service

A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

BAP1 inhibits TNMD. 2 / 2

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eidos

"These results suggest that BAP1 expression favors activation of the RhoA pathway over the Rac1 pathway , such that loss of BAP1 shifts the balance toward Rac1-driven motility and increased TEM ."

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reach

"We find that this process requires VCAM mediated adhesion between UM cells and ECs and that loss of the metastasis suppressor BAP1 enhances TEM."

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Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...