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This Service

A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

TNFAIP3 activates cell differentiation. 2 / 2

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"The deficiency of TNFAIP3 in MSCs can induce immune thrombocytopenia and influence megakaryocytic differentiation through terminating the NF-kappaB pathway that suggests TNFAIP3 play a critical role in the process of MSCs alleviate s autoimmune disease [XREF_BIBR]."

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"TNFAIP3 deficient DCs induced HDM specific T H 17cell differentiation through increased expression of the T H 17 instructing cytokines IL-1beta, IL-6, and IL-23, whereas HDM specific T H 2 cell differentiation was hampered by increased IL-12 and IL-6 production."

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Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...