IndraLab

Statements


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"As a result, overexpression of USP49 significantly inhibited cell growth of NSCLC cells."

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"USP49 was also reported to inhibit non-small cell lung cancer cell growth by suppressing PI3K/AKT signaling ."

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"In conclusion, we demonstrated that USP49 was functional in NSCLC cells, and inhibited NSCLC cell growth by suppressing PI3K and AKT signaling, suggesting that USP49 could be as a novel target for NSCLC therapy."