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This Service

A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

Reactive oxygen species activates TNFAIP3. 2 / 2

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eidos

"Our model simulation revealed that NFkB activation increased with the ROS level , which consequently increased the production of A20 ."

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eidos

"Reactive oxygen species promoted A20 expression via increased H3K4me3 modification of histones on the A20 promoter domain [ 84 ] ."

| PMC

Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...