IndraLab

Statements

G3BP1 activates USP10. 2 / 2
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"However, oxidative and nitrosative stress induce SGs formation which cause USP10 to induce ataxia-telangiectasia-mutated protein (ATM) phosphorylation which suppresses the inhibition of USP10 by G3BP1, thereby reducing ROS production and apoptosis (4, 34)."
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"Interestingly G3BP1 modulates USP10 deubiquitinase activity, as well as interacts and co-localizes with HCV NS5B protein and RNA to modulate HCV replication."
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