IndraLab

Statements



reach
"Additionally, the knockdown of USP53 suppressed proliferation, EMT, and metastasis and enhanced the paclitaxel response of TNBC cells."

reach
"Because upregulation of USP53 promoted proliferation, migration, invasion, and EMT in TNBC, we investigated whether this was due to the regulation of CRKL by USP53."

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"Taken together, USP53 promotes metastasis and EMT in TNBC."

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"Functionally, USP53 promotes TNBC cell proliferation, migration, invasion, and epithelial-mesenchymal transition (EMT)."