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Sudden Infant Death leads to the phosphorylation of SCN5A on S1503. 1 / 1
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"Using a heterologous expression system, we show that GPD1L associated with SCN5A and that the BrS- and SIDS related mutations in GPD1L caused a loss of enzymatic function resulting in glycerol-3-phosphate PKC dependent phosphorylation of SCN5A at serine 1503 (S1503) through a GPD1L dependent pathway."