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This Service

A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

RPS6KB1 phosphorylates IRS1 on tyrosine. 2 / 2

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"Thus, both AKT and MAPK inhibition may be potent to enhance melanoma chemosensitivity.Previous studies demonstrated that rapamycin inhibiting mTOR and thus p70S6K prevents the serine phosphorylation o[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"

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"These results indicate that alpha-Syn overexpression negatively regulates IRS-1 at least in part by stimulating IRS-1 Ser636 phosphorylation and tyrosine dephosphorylation by mTORC1 and S6K1."

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Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...