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A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

SRC phosphorylates AKT on T41. 1 / 1

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rlimsp

"Our data indicated that while Akt activity modulation correlated with changes in the Ser9/21 of GSK-3, changes in cSrc activity modulation using specific constitutively active and catalytically inactive mutants correlated with changes in the phosphorylation of Y216GSK-3 and Ser33/37/Thr41βcatenin, a known substrate of GSK-3, thus indicating that Src is responsible for Y216GSK-3 phosphorylation and its activation (Figure 4A and B)."

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Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...