IndraLab

Statements



sparser
"The correlation between VopZ aa 38–62 dependent inhibition of TAK1 in vitro, aa 38–62 dependent diarrhea and other intestinal pathology in vivo, and the previously described consequences of intestinal TAK1 deficiency strongly suggests that inhibition of TAK1 activation by VopZ is a critical facet of V. parahaemolyticus pathogenicity."

sparser
"The correlation between VopZ aa 38–62 dependent inhibition of TAK1 in vitro, aa 38–62 dependent diarrhea and other intestinal pathology in vivo, and the previously described consequences of intestinal TAK1 deficiency strongly suggests that inhibition of TAK1 activation by VopZ is a critical facet of V. parahaemolyticus pathogenicity."