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CACNA1E activates calcium(2+). 4 / 4
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eidos
"CACNA1E increased current density and Ca 2 + entrance , whereas calcium channel blockers inhibited NSCLC cell proliferation ."
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"The same set of mAbs was used to define the role of KChIP3-dependent Ca -mediated modulation of I underlying gain of stellate cell firing in response to repetitive stimulation (Anderson et al., 2013), and to block Kv4- and Ca -dependent EPSP boosting induced by SNX-sensitive Cav2.3 R-type Ca channels in CA1 pyramidal neurons (Figure 3) (Wang et al., 2014)."
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"Cav2.3 (R-type) voltage-activated Ca(2+) channels (VACCs), encoded by the calcium channel, voltage-dependent, R-type, α1E subunit (CACNA1E) gene, are responsible for transmission of somatic inflammatory pain, and activation of antinociception elicited by visceral inflammatory pain stimuli."
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"CACNA1E was shown to mediate a significant fraction of R-type Ca 2+ currents, specifically within the hippocampus [XREF_BIBR]."
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