IndraLab

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UCHL5 is degraded. 2 / 2

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"The overexpression of methyltransferase-like 14 (METTL14) promoted the binding of YTHDF1 to UCHL5 mRNA, which in turn inhibited the m6A methylation level of UCHL5."

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"Moreover, we found bAP15 could suppress TP53-mutant ovarian cancer cell survival by regulating TGF-beta signaling through inhibiting UCHL5 expression and dephosphorylating Smad2, consequently inducing apoptosis."

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IndraLab

Statements

UCHL5 is degraded. 2 / 2

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