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This Service

A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

STK11 bound to AMPK inhibits MTOR. 1 / 1

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"Autophagy activation can also promote tumor growth via silencing of necrosis-induced inflammatory responses, with a reciprocal response seen in tumor cells evading a necrotic fate by activating the LKB1/AMPK complex, which in turn inhibits mTOR and activates downstream autophagic processes [77]."

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Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...