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This Service

A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

Phosphorylated AKT leads to the phosphorylation of CDKN1B on S10. 1 / 1

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"Additionally, the HCC827-TM4SF5 clones not only enhanced and sustained EGFR and Akt phosphorylation but also enhanced TM4SF5 expression and p27 Kip1 Ser10 phosphorylation, which is known to cause its [MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"

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Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...