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This Service

A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

USP36 increases the amount of α-SMA. 1 / 1

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"As shown in Figures 3F,G, E-cadherin was obviously decreased and α-SMA was significantly enhanced in TECs under high glucose, while knockdown of USP36 could effectively reverse the expression of E-cadherin and α-SMA, which suggesting that up-regulation of USP36 may be an upstream mechanism for EMT and diabetic tubulointerstitial fibrosis."

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Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...