IndraLab

Statements

reach
"All these results suggested that UCHL5 knockdown repressed ox-LDL-induced excessive proliferation, migration, inflammatory response as well as synthetic phenotype switching in VMSCs.To investigate the[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
37044315
reach
"All our results suggested that UCHL5 could contribute to VSMC proliferation, migration, inflammation and phenotypic switching, thus aggravating AS progression.As a hot topic in the field of recent epi[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
37044315
reach
"Additionally, UCHL5 silencing inhibited ox-LDL-induced VSMC proliferation, migration, and inflammatory response."
38193803