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This Service

A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

USP17L2 binds cdk-4. 3 / 3

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"Moreover, increasing PI3K/Akt/mTOR activity, modulating apoptosis, altering Rho/Rac pathway would promote angiogenesis finally. xref , xref The CDK4/6‐DUB3 axis may act as an important regulatory mechanism of BCBM."

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"The interaction between CDK4/6 and DUB3 was also detected by endogenous co-immunoprecipitation and in vitro pull down (XREF_FIG)."

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"The interaction between CDK4/6 and DUB3 was also detected by endogenous co-immunoprecipitation and in vitro pull down ( xref )."

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Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...