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This Service

A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

Modified CHEK2 leads to the phosphorylation of TP53 on S20. 1 / 1

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"In vivo, ectopic expression of wild-type Chk2 and hCds1 led to increased p53 stabilization after DNA damage, whereas expression of a dominant negative Chk2 and hCds1 mutant abrogated both phosphorylation of p53 on Ser 20 and p53 stabilization."

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Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...