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This Service

A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

Recombinase inhibits USP9X. 1 / 1

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"Studies have shown that the constitutive deletion of Fam20B in mice leads to embryonic lethality at E13.5 [ xref ], and that the conditional ablation of Fam20B in the dental epithelium of mice using K14 - Cre recombinase leads to supernumerary incisors, and inactivation of mouse Fam20B by Osr2-Cre recombinase causes chondrosarcoma and postnatal ossification defects in the joint cartilage via the alterations of the WNT, BMP, and PTHrP/IHH signaling pathways [ xref , xref ]."

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Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...