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This Service

A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

EGFR bound to Galphai3 and CCDC88A activates AKT. 1 / 1

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"Later we found that GIV 's C-terminus harbors a GEF motif that binds and activates Galphai3 and upon EGF stimulation assembles an EGFR, GIV, and Galphai3 complex at the PM that prolongs association of EGFR with the PM and enhances migratory Akt signaling and cell migration."

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Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...