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USP11 activates Carcinogenesis. 2 / 2

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"Our functional results provide evidence for the crosstalk between KLF4 and USP11 in hepatic diseases; in particular, they show how USP11 enhances HCC tumorigenesis and steatosis through KLF4 inhibition."

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"Collectively, these findings suggest that USP11, as KLF4-binding partner, is an important mediator of hepatic tumorigenesis that functions via degradation of KLF4 and is a potential treatment target for liver diseases."

34114341

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IndraLab

Statements

USP11 activates Carcinogenesis. 2 / 2

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