IndraLab

Statements



reach
"All our results suggested that UCHL5 could contribute to VSMC proliferation, migration, inflammation and phenotypic switching, thus aggravating AS progression.As a hot topic in the field of recent epi[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"

reach
"Additionally, UCHL5 silencing inhibited ox-LDL-induced VSMC proliferation, migration, and inflammatory response."

reach
"Therefore, we came to the conclusion that METTL14 increased UCHL5 m 6 A level and promoted UCHL5 expression by recruiting YTHDF1, thereby enhancing VSMC proliferation, migration and phenotype switchin[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"