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This Service

A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

LPL decreases the amount of UCHL1. 2 / 2

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"Since adult (12 months, rescued) and new born LPL deficient mice (without rescue) both displayed presynaptic dysfunction (Xian et al., 2009; Liu et al., 2014), we examined adult LPL deficient mice and[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"

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"These results indicated that LPL deficiency gave rise to increased alpha-syn aggregation and decreased UCHL1 expression, which may further influence synaptic function.LPL deficient mice on C57BL/6J ba[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"

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Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...