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A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

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INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

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HIV-1 activates USP18. 4 / 4

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"Since USP18 is induced by HIV-1 in iMacs in a similar manner as MDMs , we concluded that iMacs are a better model for studying USP18 knockout / knockdown than THP-1 cells ."

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"Indeed , USP18 + / + iMacs support HIV-1 replication and USP18 is induced by HIV-1 in these cells ( Figs ."

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"We next determined if iMacs can support HIV-1 replication as previously reported 48 and if USP18 is induced by HIV-1 in iMacs ."

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"Here , we demonstrate that USP18 expression is induced by HIV-1 in a T1 IFN-dependent manner ."

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HIV-1 activates USP18. 4 / 4

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