IndraLab

Statements

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"Considering that ubiquitination is critical for both proteasomal and autophagic degradation [7, 20], we postulated that CYLD via its DUB activity interferes cardiac PQC, thereby contributing to PO-induced cardiomyopathy."
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"CYLD exaggerates pressure overload-induced cardiomyopathy via suppressing autolysosome efflux in cardiomyocytes."
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"To gain pathological insights into the CYLD-mediated cardiomyopathy towards heart failure, a larger cohort of mixed male and female littermates of adult NG and TG mice were subject to sham and TAC operations for 2 weeks."
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