IndraLab
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reach
"Moreover, the SYMPK-binding protein PRPF8 is a core component of spliceosomal complexes, which indicated that SYMPK participated in the AS regulation unbiasedly.To validate whether AS is indeed disturbed due to SYMPK deficiency, four transcripts with alterations in the splicing pattern identified by rMATS were examined by semi-quantitative RT-PCR using specific primers (Supplementary Table 1), namely, Sun1, Brdt, Meioc, and Ranbp9."