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This Service

A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

OTULIN leads to the ubiquitination of RNF31. 2 / 2

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"Supporting this, ectopic expression of an inactive OTULIN with a mutation in the PIM (Y56A) did not lead to HOIP autoubiquitination, whereas inactive OTULIN with an intact PIM led to extensive HOIP ubiquitination (XREF_FIG B)."

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"This suggested that one role of OTULIN in the LUBAC complex (XREF_FIG E) is to prevent the autoubiquitination of HOIP."

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Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...