IndraLab

Statements

PSMD14 activates epithelial to mesenchymal transition. 6 / 6
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"In esophageal squamous cell carcinoma, knockdown of PSMD14 significantly blocks SNAIL-induced epithelial mesenchymal transition (EMT), thus suppressing tumor cell migration and metastasis (Zhu et al., 2018)."
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"These findings suggest that PSMD14 could obviously induce EMT process through stabilizing SNAIL.Since SNAIL is reported to be involved in tumorigenesis and metastasis through EMT process [ 6–9 ], we p[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
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"PSMD14 is highly expressed in esophageal cancer in esophageal cancer, and inhibition of PSMD14 inhibits tumor cell migration, invasion and EMT [29]."
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"N-cadherin, E-cadherin, and vimentin are markers of EMT [ 46 ], indicating that POH1 promotes EMT in lung cancer cells by mediating the stability of Smad3."
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"Apparently, knockdown of PSMD14 suppressed SNAIL-induce EMT and tumor metastasis."
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"Functionally, PSMD14 promotes the proliferation and invasiveness of ATC cells, whereas the depletion of PSMD14 or PSMD14 inhibitor thiolutin (THL) inhibits the growth, invasiveness, and epithelial-mesenchymal transition ((EMT) of ATC cells."
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