IndraLab

Statements


5 | 2 3

sparser
"For example, USP10 interacts with G3BP2 to block p53 signal transduction, leading to poor prognosis in prostate cancer [ xref ]; USP44 promotes the development of prostate cancer by stabilizing EZH2 [ xref ], and USP2a enhances c-Myc expression via microRNA-related regulation and thus promotes tumourigenesis [ xref ]."

sparser
"The association of USP10 with G3BP2 suppresses p53 signalling which correlates to a poor prognosis in prostate cancer, highlighting an oncogenic role of USP10 in prostate cancer via G3BP2 [ xref ]."

No evidence text available

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reach
"For example, USP10 interacts with G3BP2 to block p53 signal transduction, leading to poor prognosis in prostate cancer [XREF_BIBR]; USP44 promotes the development of prostate cancer by stabilizing EZH2 [XREF_BIBR], and USP2a enhances c-Myc expression via microRNA related regulation and thus promotes tumourigenesis [XREF_BIBR]."

No evidence text available

sparser
"The N protein compromised G3BP1-USP10 and 345 G3BP1-PKR interactions (Fig. 4b, c) and disrupted G3BP2-USP10 and G3BP2-PKR interactions 346 (Fig. 4d, e) ."

reach
"Furthermore, USP10, a binding partner of G3BP2, is also an androgen responsive gene and is transcriptionally activated by the AR."

No evidence text available

No evidence text available