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This Service

A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

KCNMA1 activates GLUL. 2 / 2

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"We have found that both ROMK1 and maxi-K + expression were increased in the distal renal tubules of GS causing knockin mice on normal K + diet, suggesting that both channels are involved in renal K + wasting in GS (unpublished data)."

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"In contrast to these reports, it is noteworthy that SLO as well as human tissue LO can mediate an extensive and rapid oxidation GSH to GS in the presence of polyunsaturated fatty acids (Roy et al., 19[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"

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Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...