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A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

OTULIN leads to the phosphorylation of RELA. 2 / 2

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"Importantly, OTULIN deficiency in THP-1 monocytes led to spontaneous phosphorylation of p65 and RelA (Fig XREF_FIG A) and drastically increased turnover of IkappaBalpha (Fig XREF_FIG B), indicative of NF-kappaB activation in shOTULIN cells."

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