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A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

USP7 activates E1B-55K. 2 / 2

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"However, considering known functions of USP7 in gene expression control through regulation of histone proteins and the known relationship between adenoviral gene expression activity and the chromatinization of the adenoviral genome inside the nucleus of infected cells, it is also likely that USP7 mediated E1B-55K stability might be exerted through a mechanism other than direct deubiquitination of E1B-55K XREF_BIBR - XREF_BIBR."

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"However, during the course of this study, it became clear that USP7 not only specifically targets E1B-55K, but also exerts positive effects on other early proteins (and late proteins)."

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Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...