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This Service

A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

Mutated EGFR leads to the phosphorylation of STAT3 on tyrosine. 2 / 2

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"These results suggest that activating EGFR mutations may not directly regulate STAT3 tyrosine phosphorylation, since PC9 cells had undetectable STAT3 DNA binding activity."

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"Neither wild-type nor mutant EGFR could increase the levels of tyrosine phosphorylation of STAT3 in these cells (XREF_FIG)."

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Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...