IndraLab

Statements

UCHL5 activates Carcinoma, Endometrioid. 4 / 4
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"This study firstly reported that UCHL5 was more highly expressed on EC tissues and cell lines and UCHL5 overexpression accelerated EC growth."
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"In conclusion, our results that UCHL5 promoted the growth of EC in vivo and vitro via activating the Wnt/β-catenin signaling pathway may provide potential targets for EC control in the future."
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"Lastly, in endometrial cancer, UCHL5 accelerates the growth of EC via activating the Wnt/β-Catenin signaling pathway that could be an attractive target for EC treatment [ 21 ]."
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"In conclusion, UCHL5 accelerated the growth of EC via the Wnt/beta-catenin pathway and was expected to be an attractive target for EC treatment."
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