Other Services

This Service

A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

AHR leads to the ubiquitination of RAC1. 1 / 1

| 1

➶

reach

"Taken together, our data suggest that AhR activation by BaP induces JNK dependent RAC1 ubiquitination, which inhibits the RAC1-PI3K-AKT-signaling axis after TLR4 activation and thus potentially contributes to the AhR dependent attenuation of pro inflammatory signaling in a non genomic manner (XREF_FIG)."

33868237

Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...