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A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

Histone methylates TP53. 3 / 4

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"Moreover, G9a and HDAC1 epigenetically repress p53 target genes by maintaining H3K9 methylation and histone deacetylation of the p53 target genes promoter 41."

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"Expression of a constitutively active prolactin receptor causes histone trimethylation of the p53 gene in breast cancer."

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"Histone trimethylation of the p53 gene by expression of a constitutively active prolactin receptor in prostate cancer cells."

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INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...

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IndraLab

Statements

Histone methylates TP53. 3 / 4

Our Sources:

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