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This Service

A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

AKT1 leads to the phosphorylation of RPS6KB1-S6K. 1 / 1

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"Importantly, UV induced increases in p70 (S6k) phosphorylation at Thr (389) and Thr (421)/Ser (424) were dramatically inhibited by pretreatment of cells with rapamycin, LY294002, or PD98059, whereas overexpression of dominant negative mutants of PKClambda and iota and Akt1 did not inhibit p70 (S6k) phosphorylation at Thr (389) and Thr (421)/Ser (424)."

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Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...