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This Service

A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

HCN1 inhibits PEX5L. 2 / 2

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"Moreover, deletion of the extreme N-terminus of HCN1 strongly enhances the ability of TRIP8b (1a) to downregulate channel surface expression (A. Kushnir and B. Santoro, unpublished observation)."

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"Truncation of the entire extreme C-terminus of HCN1 distal to the CNBD (HCN1 DeltaCX) also fully blocked the capacity of TRIP8b (1a) to decrease channel surface expression with no effect on the enhancement of channel surface expression with TRIP8b (1a-4), similar to the effects seen above with the SNL deletion (XREF_FIG)."

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Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...