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A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

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INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

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SRC increases the amount of CCND1. 3 / 8

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"These results indicate that progestin induction of CCND1 expression mediated by activation of Src and MAPK and PI3K signaling pathways plays an important role in mediating the effect of progesterone o[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"

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"Furthermore, suppression of Src activity using the PP2 inhibitor abolished p120 and p130CAS phosphorylation and also reversed the increased expression of SNAI1, CCND1, MYC and cadherin 11 after PLEKHA7 knockdown (XREF_FIG and XREF_SUPPLEMENTARY)."

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"Inhibition of the Src and Akt pathway would reduce Ccnd1 expression."

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INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...

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Statements

SRC increases the amount of CCND1. 3 / 8

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