IndraLab

Statements

Glu-Ala inhibits RELA. 4 / 4
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"EA at Zusanli could inhibit NF-kappaBp65 and proinflammatory cytokines production after intestinal I/R injury; its mechanism may be related to the cholinergic anti-inflammatory pathway."
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"EA treatment significantly reduced the nuclear p65 content, accompanied by a decreased ratio of p65 protein content in the nucleus to that in the cytoplasm in the adipose tissue."
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"Furthermore, immunofluorescence staining also confirmed that 2Hz EA obviously suppressed the co-expression of microglia activation marker CD11b and TLR4 or MyD88, as well as the activation of NF-kappaB p65 in SNI rats."
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"This study just attempts to explore its possible mechanism from the perspective of cholinergic anti-inflammatory pathway, and the results showed that both EA and alpha7 nAChR agonists could relieve the intestine damage and decrease levels of mucosal NF-kappaBp65 and serum IL-6 and TNF-alpha."
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