IndraLab

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Infections increases the amount of TNFAIP3. 7 / 7
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"Both OSU and NCDV NSP1 interact with β-TrCP, indicating that an association between NSP1 and target is likely necessary to induce turnover, as is true for NSP1 proteins that mediate IRF3 degradation.Recently, microarray analysis of host gene expression in HT-29 (human colorectal adenocarcinoma) cells has demonstrated that infection with SA11-4F, Wa, or bovine A5-13 RVA upregulates transcription of the tumor necrosis factor (TNF)-α-induced protein 3 (TNFAIP3) gene (Bagchi et al., 2012)."
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"qRT-PCR resulted showed that ETEC infection significantly increased the expression of CCL20, CXCL2, CXCL8, AP-1, IL-1, IL-6, IL-11, NFKBIA, MAP3K12, MAPK15, PTGS2, and TNFAIP3, which pretreatment with L. reuteri inhibited these increases."
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"FIG 5: Deltacoronavirus infection synchronously activates endogenous circTNFAIP3 and TNFAIP3 expression."
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"(A) Expression of TNFAIP3 pre-mRNA, mRNA, and circRNA induced by PDCoV infection."
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"found that Mtb infection could enhance the expression of TNFAIP3, which attenuated the inflammatory signaling and facilitated the survival of Mtb bacterial."
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"Therefore, we performed experiments using cell lines A549 and THP1, and observed that IAV infection remarkably upregulated the expression of Tnfaip3 (Supplementary Figures 6C, D)."
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"The TNFAIP3 hub-central gene is a central regulator of immunopathology because it is a key player in the negative feedback regulation of the NF-κB signaling pathway (Vereecke et al., 2009), and increased of TNFAIP3 expression levels in M. bovis-infected animals modulates the host immune response and decreases proinflammatory cytokines (especially TNF) by inhibition NF-κB signaling, thereby leads to progression of M. bovis infection (Kumar et al., 2015)."
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