IndraLab
Statements
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                                  "In particular, one of these mechanisms involves a dynamic regulation of the interaction between MEF2C and the transcriptional co-repressors class IIa histone deacetylases (HDACs) such as HDAC4 and −5: in proliferating myoblasts MEF2C interacts with HDACs and its activity is repressed, and myogenic signals disrupt this interaction, thereby alleviating the repression of MEF2C activity xref  xref  xref  xref  xref ."