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A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

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Statements

USP18 inhibits Chemokine. 2 / 2

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"Further studies show that the expression of USP18 by beta islet cells themselves is important for inhibiting diabetes.75, 76 On the one hand, the upregulation of USP18 expression by IFN-I in beta islet cells prevents the activity of proinflammatory chemokines such as CCL5, CXCL10, and IL-15 and consequently inhibits insulitis."

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"75, 76 On the one hand, the upregulation of USP18 expression by IFN-I in beta islet cells prevents the activity of proinflammatory chemokines such as CCL5, CXCL10, and IL-15 and consequently inhibits insulitis."

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INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...