IndraLab

Statements

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"miR-29c targets TNFAIP3, inhibits cell proliferation and induces apoptosis in hepatitis B virus related hepatocellular carcinoma."
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"HIV-1 subsets upregulated CXCR4 and TNFAIP3, which modulate cell proliferation and initiate inflammatory immune responses, respectively."
35043114
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"31 In melanoma, the orchestrated deletion of TNFAIP3 significantly diminishes melanoma cell proliferation and tumour growth in mouse models."
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"However, when overexpressing TNFAIP3 and PLAU or upon activation of the NFkappaB pathway, all the inhibitory effects of SFE were reversed (XREF_FIG A-D), suggesting that SFE blocked the p65 promotion of TNFAIP3 and PLAU expression to inhibit ESCC cell proliferation and metastasis."
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"TNFAIP3 in MSCs inhibits TNF-α generation and promotes IL-10 proliferation; whereas knockdown of TNFAIP3 weakens the immunosuppressive capacity of MSCs both in vitro and in vivo as indicated that inhibition of T-cell proliferation is reversed."
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"Additionally, the overexpression of TNFAIP3 enhances the proliferation, migration and invasion of KYSE150‐TRCs, confirming our earlier speculation that TNFAIP3 is the key molecule responsible for the growth‐inhibitory effect of ZSH‐2208 on ESCC‐TRCs.3 DISCUSSION."
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"Although T cells from both diseases express high levels of genes encoding for inflammatory proteins and chemokines, COVID-19 subsets expressed high levels of GZMB and CXC3CR1 (Figure 5E), suggesting increased cytotoxicity and terminal effector function, while HIV-1 subsets showed upregulation of CXCR4 and TNFAIP3, which modulate cell proliferation and initiate inflammatory immune responses, respectively."
36992700
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"In addition, the knockdown of TNFAIP3 restores the significant decrease in invasion and proliferation in miR-605-5p-inhibitor-transfected lung cancer cells."
31452243
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"We also demonstrate that knockout (KO) of TNFAIP3 blocks FGFR1 signaling-promoted DCIS cell proliferation and progression, suggesting that TNFAIP3 is required for FGFR1 signaling-promoted DCIS growth and progression."
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"Cluster 2 contained IL8, IL1A, PTGS2, DTR, TNFAIP3, and CXCL3 that were up-regulated and linked primarily to inflammatory response and cell proliferation."
16330358
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"TNFAIP3 deficient B-cells are hyper-reactive to antigen stimulation, leading to enhanced proliferation and survival."
27248180
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"Similarly, the knockdown of YTHDF2′ and TNFAIP3 increased the viability and proliferation of T98G/TR cells compared with YTHDF2′ knockdown alone, whereas the JSH‐23 subjection decreased the level (Supplementary figure 3d, e)."
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"Mutations in TNFRSF14, CTSS, and TNFAIP3 recruit more Tfh cells to support FL proliferation."
37063433
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"Both Emu- BCL10 and Emu- API2-MALT1 mice develop splenic marginal zone hyperplasia but not lymphoma [86,113], while TNFAIP3 (A20) deficiency in B-cells enhances B-cell proliferation with an excessive [MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
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