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This Service

A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

SRC leads to the phosphorylation of AKT1. 2 / 2

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"PI3K and AKT kinase inhibitor Ly294-002, Syc kinase inhibitor Bay61, Src kinase inhibitor PP2, but not the respective control PP3, also specifically blocked AKT1 phosphorylation in human platelets induced by CAPs at very low concentrations (XREF_FIG)."

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"In addition, CD99 induced c-Src inactivation may inhibit the tyrosyl phosphorylation of Akt1, which is a prerequisite for Akt1 activation [50,64]."

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Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...