A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

IndraLab

Statements

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phosphosite cbn pc11 biopax bel_lc signor biogrid tas lincs_drug hprd trrust | geneways tees isi trips rlimsp medscan sparser reach
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"Together, our results demonstrate that TSLP promotes Th9 cell differentiation and function and define a requirement for IL-9 in TSLP induced allergic inflammation."
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"IL-9 promotes inflammation by stimulating growth of hematopoietic cells, particularly mast cells, and the secretion of factors including chemokines that recruit additional cells to inflamed sites."
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"Furthermore, transgenic overexpression of IL-9 in lung induced severe allergic inflammation and asthma 52."
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"IL-9 expressing ILC2 cells have also been observed in a papain induced lung inflammation model; however, this expression was transient and transitioned to IL-5 and/or IL-13 expression over time."
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"IL-9 contributes to the development of psoriatic lesions, by inducing Th17 related inflammation and by promoting angiogenesis [XREF_BIBR]."
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"Roles of IL-9 in S. japonicum infection induced hepatic granulomatous and fibrosing inflammation."
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"In these patients, IL-9 levels correlated with clinical severity, IgE levels, and CCL17 levels, indicating an intimate relationship of IL-9 with classical mediators of T H 2 induced inflammation [XREF_BIBR]."
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"A number of studies showed that IL-9 has anti-inflammatory properties, whereas other studies found that IL-9 effectively promoted inflammation."
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"IL-9 and mucosal MCs contributed to barrier function loss, dissemination, and inflammation in experimental models of impaired intestinal barrier function and were upregulated in patients with CD."
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"Th2 cells generate cytokines such as IL-4, IL-5, IL-6, IL-9 and IL-13, which mediate allergic inflammation."
IL9 activates inflammatory response. 1 / 1
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"IL-9 is also implicated in playing a role in airway smooth muscle inflammation, and asthma [16]."